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![El-deiry et al 1992 nature genetics guide: >> http://sts.cloudz.pw/download?file=el-deiry+et+al+1992+nature+genetics+guide << (Download)
El-deiry et al 1992 nature genetics guide: >> http://sts.cloudz.pw/read?file=el-deiry+et+al+1992+nature+genetics+guide << (Read Online)
Letter. Nature Genetics 14, 482 - 486 (1996) doi:10.1038/ng1296-482. Identification of BTG2, an antiproliferative p53?dependent component of the DNA damage cellular response pathway. Jean-Pierre Rouault1, 2, *, Nicole Falette1, 3, *, Fabienne Guehenneux1, *, Celine Guillot1, Ruth Rimokh1, Qing Wang3, Cyril
Also, the cyclin-dependent kinase inhibitor p21/waf1 has a well-established role in the p53-induced G1 arrest of the cell cycle (El-Deiry et al., 1993). Differential display was performed as described (Murphy et al., 1996) on the murine embryo fibroblast cell line Val5, which expresses a temperature-sensitive p53 protein
Nature Genetics 16, 298 - 302 (1997) doi:10.1038/ng0797-298 Cell 69, 1237?1245 (1992). | Article | PubMed | ISI | ChemPort |; Gu, Y., Turek, PubMed |; Dulic, V. et al. p53-dependent inhibition of cyclin-dependent kinase-activities in human fibroblasts during radiation-induced G1 arrest. Cell 76, 1013?1023 (1994).
The p53 tumour suppressor promotes cell-cycle arrest or apoptosis in response to cellular stress, such as DNA damage and oncogenesis. This role of p53 is important for its tumour-suppression function and depends, at least in part, on its ability to bind to specific DNA sequences and activate the transcription of target genes
2 Jul 1992 El-Deiry, W. S., Kern, S. E., Pietenpol, J. A., Kinzler, K. W. & Vogelstein, B. Nature Genetics 1, 45–49 (1992). Show context. 9. 18. Yonish, R. E. et al. Nature 353, 345–347 (1991). Show context. Article. 19. Donehower, L. A. et al. Nature 356, 215–221 (1992). Show context. CAS · PubMed · Article. 20.
2 Jul 1992 Momand, J., Zambetti, G. P., Olson, D. C., George, D. L. & Levine, A. J. Cell 69, 1237–1245 (1992). Show context. View ref 2 on CAS El-Deiry, W. S., Kern, S. E., Pietenpol, J. A., Kinzler, K. W. & Vogelstein, B. Nature Genetics 1, 45–49 (1992). Show context. 9. Donehower, L. A. et al. Nature 356, 215–221
Johnson, P. & Benchimol, S. Friend virus induced murine erythroleukaemia: The p53 locus, in Cancer Surveys 12, (ed. A.J. Levine) 137?151 (Cold Spring Harbor Press, New York, 1992). | ChemPort |; Baker, S.J. et al. Chromosome 17 deletions and p53 gene mutations in colorectal carcinoma. Science 244, 217?221 (1989)
Definition of a consensus binding site for p53. Wafik S. El-Deiry; , Scott E. Kern; , Jennifer A. Pietenpol; , Kenneth W. Kinzler; [] & Bert Vogelstein; - Show fewer authors. Nature Genetics 1, 45–49 (1992); doi :10.1038/ng0492-45; Download Citation. Received: 10 January 1992; Accepted: 24 February 1992; Published
et al., 1992; El-Deiry et al., 1992). Sequence specific transactivation (SST) by p53 has been shown to be dependent upon three major independent domains: (i) a central core domain responsible for sequence-specific DNA binding (amino acid residues 102 – 292) (Bargonetti et al., 1993; Pavletich et al., 1993; Cho et al.,
K-ras and p53 gene mutations in pancreatic cancer: ductal and nond](http://cdn08.dayviews.com/501/_u4/_u1/_u9/_u3/_u0/_u3/u4193030/96811_1518605379/El_deiry_et_al_1992_nature_genetics_guide_http_sts_cloudz_pw_download_file_el_deiry_et_al_1992.jpg)
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El-deiry et al 1992 nature genetics guide: >> http://sts.cloudz.pw/download?file=el-deiry+et+al+1992+nature+genetics+guide << (Download)
El-deiry et al 1992 nature genetics guide: >> http://sts.cloudz.pw/read?file=el-deiry+et+al+1992+nature+genetics+guide << (Read Online)
Letter. Nature Genetics 14, 482 - 486 (1996) doi:10.1038/ng1296-482. Identification of BTG2, an antiproliferative p53?dependent component of the DNA damage cellular response pathway. Jean-Pierre Rouault1, 2, *, Nicole Falette1, 3, *, Fabienne Guehenneux1, *, Celine Guillot1, Ruth Rimokh1, Qing Wang3, Cyril
Also, the cyclin-dependent kinase inhibitor p21/waf1 has a well-established role in the p53-induced G1 arrest of the cell cycle (El-Deiry et al., 1993). Differential display was performed as described (Murphy et al., 1996) on the murine embryo fibroblast cell line Val5, which expresses a temperature-sensitive p53 protein
Nature Genetics 16, 298 - 302 (1997) doi:10.1038/ng0797-298 Cell 69, 1237?1245 (1992). | Article | PubMed | ISI | ChemPort |; Gu, Y., Turek, PubMed |; Dulic, V. et al. p53-dependent inhibition of cyclin-dependent kinase-activities in human fibroblasts during radiation-induced G1 arrest. Cell 76, 1013?1023 (1994).
The p53 tumour suppressor promotes cell-cycle arrest or apoptosis in response to cellular stress, such as DNA damage and oncogenesis. This role of p53 is important for its tumour-suppression function and depends, at least in part, on its ability to bind to specific DNA sequences and activate the transcription of target genes
2 Jul 1992 El-Deiry, W. S., Kern, S. E., Pietenpol, J. A., Kinzler, K. W. & Vogelstein, B. Nature Genetics 1, 45–49 (1992). Show context. 9. 18. Yonish, R. E. et al. Nature 353, 345–347 (1991). Show context. Article. 19. Donehower, L. A. et al. Nature 356, 215–221 (1992). Show context. CAS · PubMed · Article. 20.
2 Jul 1992 Momand, J., Zambetti, G. P., Olson, D. C., George, D. L. & Levine, A. J. Cell 69, 1237–1245 (1992). Show context. View ref 2 on CAS El-Deiry, W. S., Kern, S. E., Pietenpol, J. A., Kinzler, K. W. & Vogelstein, B. Nature Genetics 1, 45–49 (1992). Show context. 9. Donehower, L. A. et al. Nature 356, 215–221
Johnson, P. & Benchimol, S. Friend virus induced murine erythroleukaemia: The p53 locus, in Cancer Surveys 12, (ed. A.J. Levine) 137?151 (Cold Spring Harbor Press, New York, 1992). | ChemPort |; Baker, S.J. et al. Chromosome 17 deletions and p53 gene mutations in colorectal carcinoma. Science 244, 217?221 (1989)
Definition of a consensus binding site for p53. Wafik S. El-Deiry; , Scott E. Kern; , Jennifer A. Pietenpol; , Kenneth W. Kinzler; [] & Bert Vogelstein; - Show fewer authors. Nature Genetics 1, 45–49 (1992); doi :10.1038/ng0492-45; Download Citation. Received: 10 January 1992; Accepted: 24 February 1992; Published
et al., 1992; El-Deiry et al., 1992). Sequence specific transactivation (SST) by p53 has been shown to be dependent upon three major independent domains: (i) a central core domain responsible for sequence-specific DNA binding (amino acid residues 102 – 292) (Bargonetti et al., 1993; Pavletich et al., 1993; Cho et al.,
K-ras and p53 gene mutations in pancreatic cancer: ductal and nonductal tumors progress through different genetic lesions. Cancer Res. 54, 1556?1560 (1994). | PubMed | ISI | ChemPort |; Redston, M.S. et al. p53 mutations in pancreatic carcinoma and evidence of common involvement of homocopoiymer tracts in DNA
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